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In mouse models of androgenetic alopecia, topical GHK-Cu has produced hair regrowth comparable to or modestly better than 5 percent minoxidil, with faster entry into anagen phase

It is available as a research peptide or cosmetic ingredient, but clinical use remains investigational and unsupported by large human trials

Another potential mechanism could be the role of NAD + consuming PARP enzymes which are involved in repairing DNA damage, which could be induced by chemotherapy

instead, they work in fundamentally different ways

Phase 3 trials of tirzepatide did not exclude participants taking antioxidant supplements, and subgroup analyses showed no difference in adverse events or efficacy between supplement users and non-users

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